• Home
  • About Journal
  • Editorial Board
  • Instruction
  • Subscription
  • Journal Online
  • Advertisement
  • Contact Us
  • 中文
  • Forthcoming Articles
  • Current Issue
  • Archive
  • Download Articles
  • Read Articles
  • Most Cited Articles

Prevention and Treatment of Cardiovascular Disease ›› 2022, Vol. 12 ›› Issue (6): 91-94.

Previous Articles    

  

  • Online:2022-02-25 Published:2022-06-10

PDF (PC)

65

Knowledge

0

Abstract

Cite this article

share this article

0
    /   /   Recommend

Add to citation manager EndNote|Reference Manager|ProCite|BibTeX|RefWorks

URL: https://www.xxgbfzzs.com/EN/

         https://www.xxgbfzzs.com/EN/Y2022/V12/I6/91

References

[1] 中国心血管健康与疾病报告2020概要[J].中国循环杂志,2021,36(6):521-545.
[2] Zhu Y, Xian X, Wang Z, et al.Research Progress on the Relationship between Atherosclerosis and Inflammation[J]. Biomolecules,2018,8(3):80.
[3] Winkels H,Ehinger E,Ghosheh Y,et al.Atherosclerosis in the single-cell era.[J].Curr Opin Lipidol,2018,5:389-396.
[4] Selathurai A,Deswaerte V,Kanellakis P,et al.Natural killer (NK) cells augment atherosclerosis by cytotoxic-dependent mechanisms.[J].Cardiovasc Res,2014,1:128-137.
[5] Souilhol C,Harmsen MC,Evans PC,et al.Endothelial-mesenchymal transition in atherosclerosis.[J].Cardiovasc Res,2018,4:565-577.
[6] Simionescu M.Implications of early structural-functional changes in the endothelium for vascular disease.[J].Arterioscler Thromb Vasc Biol,2007,2:266-274.
[7] Crea F,Libby P.Acute Coronary Syndromes: The Way Forward From Mechanisms to Precision Treatment.[J].Circulation,2017,12:1155-1166.
[8] Zhou J,Li YS,Chien S.Shear stress-initiated signaling and its regulation of endothelial function.[J].Arterioscler Thromb Vasc Biol,2014,10:2191-2198.
[9] Lluberas N, Trías N, Brugnini A, et al.Lymphocyte subpopulations in myocardial infarction: a comparison between peripheral and intracoronary blood[J].Springerplus. 2015,4:744.
[10] Theodorou K,Boon RA.Endothelial Cell Metabolism in Atherosclerosis[J].Front Cell Dev Biol,2022,:82.
[11] Nahrendorf M,Swirski FK,Aikawa E,et al.The healing myocardium sequentially mobilizes two monocyte subsets with divergent and complementary functions.[J].J Exp Med,2007,12:3037-3047.
[12] Mentz RJ,Bakris GL,Waeber B,et al.The past, present and future of renin-angiotensin aldosterone system inhibition.[J].Int J Cardiol,2013,5:1677-1687.
[13] Kumric M,Kurir TT,Borovac JA,et al.The Role of Natural Killer (NK) Cells in Acute Coronary Syndrome: A Comprehensive Review[J].Biomolecules,1970,11.
[14] Ong SB,Hernández-Reséndiz S,Crespo-Avilan GE,et al.Inflammation following acute myocardial infarction: Multiple players, dynamic roles, and novel therapeutic opportunities.[J].Pharmacol Ther,2018:73-87.
[15] Wang X,Guo Z,Ding Z,et al.Inflammation, Autophagy, and Apoptosis After Myocardial Infarction.[J].J Am Heart Assoc,2018,9.
[16] Winkels H,Ley K.Natural Killer Cells at Ease: Atherosclerosis Is Not Affected by Genetic Depletion or Hyperactivation of Natural Killer Cells.[J].Circ Res,2018,1:6-7.
[17] Engelbertsen D,Autio A,Verwilligen RAF,et al.Increased lymphocyte activation and atherosclerosis in CD47-deficient mice.[J].Sci Rep,2019,1:10608.
[18] Knorr M,Münzel T,Wenzel P.Interplay of NK cells and monocytes in vascular inflammation and myocardial infarction.[J].Front Physiol,2014:295.
[19] Bonaccorsi I,Spinelli D,Cantoni C,et al.Symptomatic Carotid Atherosclerotic Plaques Are Associated With Increased Infiltration of Natural Killer (NK) Cells and Higher Serum Levels of NK Activating Receptor Ligands.[J].Front Immunol,2022:1503.
[20] Nour-Eldine W,Joffre J,Zibara K,et al.Genetic Depletion or Hyperresponsiveness of Natural Killer Cells Do Not Affect Atherosclerosis Development.[J].Circ Res,2018,1:47-57.
[21] Jonasson L,Backteman K,Ernerudh J.Loss of natural killer cell activity in patients with coronary artery disease.[J].Atherosclerosis,2005,2:316-21.
[22] Laskarin G,Persic V,Ruzic A,et al.Perforin-mediated cytotoxicity in non-ST elevation myocardial infarction.[J].Scand J Immunol,2011,2:195-204.
[23] Backteman K,Ernerudh J,Jonasson L.Natural killer (NK) cell deficit in coronary artery disease: no aberrations in phenotype but sustained reduction of NK cells is associated with low-grade inflammation.[J].Clin Exp Immunol,2014,1:104-112.
[24] Hong YJ,Cho YN,Kim TJ,et al.Functional deficiency of natural killer cells in acute coronary syndrome is related to ineffective degranulation.[J].Int J Cardiol,2014,3:613-615.
[25] Kumric M,Kurir TT,Borovac JA,et al.The Role of Natural Killer (NK) Cells in Acute Coronary Syndrome: A Comprehensive Review.[J].Biomolecules,1970,11.
[26] Aranha FC, Ribeiro U Jr, Basse P, et al.Interleukin-2-activated natural killer cells may have a direct role in the control of Leishmania (Leishmania) amazonensis promastigote and macrophage infection[J]. Scandinavian journal of immunology, 2005, 62(4): 334-341.
[27] Ayach BB,Yoshimitsu M,Dawood F,et al.Stem cell factor receptor induces progenitor and natural killer cell-mediated cardiac survival and repair after myocardial infarction.[J].Proc Natl Acad Sci U S A,2006,7:2304-2309.
[28] Bouchentouf M,Williams P,Forner KA,et al.Interleukin-2 enhances angiogenesis and preserves cardiac function following myocardial infarction.[J].Cytokine,2011,3:732-738.
[29] Yan W,Song Y,Zhou L,et al.Immune Cell Repertoire and Their Mediators in Patients with Acute Myocardial Infarction or Stable Angina Pectoris.[J].Int J Med Sci,2017,2:181-190.
[30] Matsumoto K,Obana M,Kobayashi A,et al.Blockade of NKG2D/NKG2D ligand interaction attenuated cardiac remodelling after myocardial infarction.[J].Cardiovasc Res,2019,4:765-775.
[31] Wang J,Chen Z,Dai Q,et al.Intravenously delivered mesenchymal stem cells prevent microvascular obstruction formation after myocardial ischemia/reperfusion injury[J].Basic Res Cardiol,2020,4:40.

Related Articles 0

No related articles found!

Recommended Articles

Metrics

Viewed
Full text


Abstract

Comments

  • Abstract
  • References
  • Related Articles
  • Recommended Articles
  • Metrics
  • Comments
TOP
Copyright © 2020 Prevention and Treatment of Cardiovascular Disease, All Rights Reserved.
Tel: 020-83868176 Fax: 020-83868176  Powered by Beijing Magtech Co. Ltd